| 1 |
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Show voter details
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moderate
|
Proxies\__CG__\App\Entity\Entry {#2222
+user: Proxies\__CG__\App\Entity\User {#2873 …}
+magazine: Proxies\__CG__\App\Entity\Magazine {#1550 …}
+image: null
+domain: Proxies\__CG__\App\Entity\Domain {#2875 …}
+slug: "What-are-some-small-things-we-should-change-about-the"
+title: "What are some small things we should change about the human body?"
+url: null
+body: """
Ripping off a post I just saw in the Isaac Arthur subreddit. Imagining we work out the technical ability. Examples they suggested were:\n
\n
- No periods\n
- No balding\n
- No nausea\n
- No body odor\n
- Resistance to obesity and muscle wasting
"""
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date: 2024-10-11 21:35:30.0 +02:00
}
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1 => App\Entity\EntryComment {#2218
+user: App\Entity\User {#260 …}
+entry: Proxies\__CG__\App\Entity\Entry {#2222 …2}
+magazine: Proxies\__CG__\App\Entity\Magazine {#1550 …}
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+parent: Proxies\__CG__\App\Entity\EntryComment {#2221 …}
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+body: "Only for half of protein and carbohydrates that can go into the gluconeogenesis pathway. That'll increase uti incidence, shred your kidneys with proteinurea, and lead to ketoacidosis when you're dealing with the other half of the proteins and all the other lipids, which before it kills you will allow you to excrete that waste from you lungs while you smell like you've been huffing nail polish remover. I think the out the butt option is generally preferable."
+lang: "en"
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date: 2024-01-03 18:39:49.0 +01:00
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"@LesserAbe@lemmy.world"
"@gibmiser@lemmy.world"
"@roguetrick@kbin.social"
"@theneverfox@pawb.social"
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"@LesserAbe@lemmy.world"
"@gibmiser@lemmy.world"
"@roguetrick@kbin.social"
"@theneverfox@pawb.social"
]
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-id: 277034
-bodyTs: "'allow':51 'butt':76 'carbohydr':7 'deal':32 'excret':54 'general':79 'gluconeogenesi':13 'go':10 'half':3,36 'huf':67 'incid':19 'increas':17 'ketoacidosi':28 'kidney':22 'kill':48 'lead':26 'like':63 'lipid':44 'll':16 'lung':59 'nail':68 'option':77 'pathway':14 'polish':69 'prefer':80 'protein':5,39 'proteinurea':24 're':31 'remov':70 'shred':20 'smell':62 'think':72 'uti':18 've':65 'wast':56"
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+editedAt: DateTimeImmutable @1711295080 {#2261
date: 2024-03-24 16:44:40.0 +01:00
}
+createdAt: DateTimeImmutable @1704303589 {#2220
date: 2024-01-03 18:39:49.0 +01:00
}
+"title": 277034
}
2 => App\Entity\EntryComment {#2262
+user: App\Entity\User {#260 …}
+entry: Proxies\__CG__\App\Entity\Entry {#2222 …2}
+magazine: Proxies\__CG__\App\Entity\Magazine {#1550 …}
+image: null
+parent: Proxies\__CG__\App\Entity\EntryComment {#2263 …}
+root: Proxies\__CG__\App\Entity\EntryComment {#2219 …}
+body: """
That's a complicated question, because yes there are weight loss drugs that mimic certain aspects of diabetes, but through different mechanisms of action. A good example is a SGLT2 inhibitor medication, which explicitly creates glycosuria in a non-diabetic person, which is kind of a defining feature of diabetes mellitus. While that's a generally very effective drug and should be used more for obesity control, it will also increase incidence of UTI and (due to a loss of sugar and thus forcing metabolism of fats/proteins) ketoacidosis.\n
\n
The insulin/glucagon relationship is why diabetic ketoacidosis only shows up in type 1 diabetics and not type 2 (they get what's called hyperosmolar hyperglycemic state), and is an important part of liver fat production. Ketoacidosis is generally associated with that but is also present in starvation and alcoholics that don't eat actual food.\n
\n
The greater point I was making wasn't about carbohydrates turning into fats. It's that most fats and about half of proteins cannot be turned into carbohydrates. They must be metabolized into ketones and then acetone. If you absorb them and cannot store them like in the OP's hypothetical, you'll end up having to immediately metabolize them and will end up with ketoacidosis.
"""
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date: 2024-01-04 18:23:02.0 +01:00
}
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"@LesserAbe@lemmy.world"
"@gibmiser@lemmy.world"
"@roguetrick@kbin.social"
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"@LesserAbe@lemmy.world"
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date: 2024-04-02 15:53:56.0 +02:00
}
+createdAt: DateTimeImmutable @1704388982 {#2265
date: 2024-01-04 18:23:02.0 +01:00
}
+"title": 280094
}
0 => App\Entity\EntryComment {#2208
+user: App\Entity\User {#260 …}
+entry: Proxies\__CG__\App\Entity\Entry {#2222 …2}
+magazine: Proxies\__CG__\App\Entity\Magazine {#1550 …}
+image: null
+parent: Proxies\__CG__\App\Entity\EntryComment {#2209 …}
+root: Proxies\__CG__\App\Entity\EntryComment {#2219 …}
+body: """
It's all about degree. Essentially what I'm talking about is severe dysregulation of lipid/protein metabolism that is generally only seen in type 1 diabetics, but it's the natural extension of extreme lipid and protein metabolism. I'm not familiar with that supplement, however.\n
\n
As far as you watching yourself for it, don't worry, you'd notice being acidotic. I've seen it plenty of times at the hospital and you'd be huffing and puffing the acid off with terrible stomach pain and be generally having a bad day. While that supplement is associated with urine acetone increases, it's not quite the degree that I'm taking about.\n
\n
Essentially, inhibiting fat production and storage and eliminating it result in very different effects. Insulin, by the way, is a very important mediator in fat production and storage. It's why so many new diagnosis type 1 diabetic children are often underweight.
"""
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date: 2024-01-04 08:45:01.0 +01:00
}
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"@LesserAbe@lemmy.world"
"@gibmiser@lemmy.world"
"@roguetrick@kbin.social"
"@theneverfox@pawb.social"
"@LesserAbe@lemmy.world"
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+children: Doctrine\ORM\PersistentCollection {#2204 …}
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-id: 278949
-bodyTs: "'1':25,150 'aceton':101 'acid':81 'acidot':62 'associ':98 'bad':92 'children':152 'd':59,75 'day':93 'degre':5,108 'diabet':26,151 'diagnosi':148 'differ':126 'dysregul':14 'effect':127 'elimin':121 'essenti':6,114 'extens':32 'extrem':34 'familiar':42 'far':48 'fat':116,138 'general':20,89 'hospit':72 'howev':46 'huf':77 'import':135 'increas':102 'inhibit':115 'insulin':128 'lipid':35 'lipid/protein':16 'm':9,40,111 'mani':146 'mediat':136 'metabol':17,38 'natur':31 'new':147 'notic':60 'often':154 'pain':86 'plenti':67 'product':117,139 'protein':37 'puf':79 'quit':106 'result':123 'seen':22,65 'sever':13 'stomach':85 'storag':119,141 'supplement':45,96 'take':112 'talk':10 'terribl':84 'time':69 'type':24,149 'underweight':155 'urin':100 've':64 'watch':51 'way':131 'worri':57"
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date: 2024-03-27 03:44:32.0 +01:00
}
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date: 2024-01-04 08:45:01.0 +01:00
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}
]
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-bodyTs: "'abil':19 'arthur':11 'bald':27 'bodi':31 'exampl':20 'imagin':13 'isaac':10 'muscl':37 'nausea':29 'obes':35 'odor':32 'period':25 'post':4 'resist':33 'rip':1 'saw':7 'subreddit':12 'suggest':22 'technic':18 'wast':38 'work':15"
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date: 2024-03-21 03:39:54.0 +01:00
}
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date: 2024-01-02 20:40:53.0 +01:00
}
+__isInitialized__: true
…2
} |
|
Show voter details
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|
edit
|
Proxies\__CG__\App\Entity\Entry {#2222
+user: Proxies\__CG__\App\Entity\User {#2873 …}
+magazine: Proxies\__CG__\App\Entity\Magazine {#1550 …}
+image: null
+domain: Proxies\__CG__\App\Entity\Domain {#2875 …}
+slug: "What-are-some-small-things-we-should-change-about-the"
+title: "What are some small things we should change about the human body?"
+url: null
+body: """
Ripping off a post I just saw in the Isaac Arthur subreddit. Imagining we work out the technical ability. Examples they suggested were:\n
\n
- No periods\n
- No balding\n
- No nausea\n
- No body odor\n
- Resistance to obesity and muscle wasting
"""
+type: "article"
+lang: "en"
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date: 2024-10-11 21:35:30.0 +02:00
}
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+badges: Doctrine\ORM\PersistentCollection {#2886 …}
+children: [
1 => App\Entity\EntryComment {#2218
+user: App\Entity\User {#260 …}
+entry: Proxies\__CG__\App\Entity\Entry {#2222 …2}
+magazine: Proxies\__CG__\App\Entity\Magazine {#1550 …}
+image: null
+parent: Proxies\__CG__\App\Entity\EntryComment {#2221 …}
+root: Proxies\__CG__\App\Entity\EntryComment {#2219 …}
+body: "Only for half of protein and carbohydrates that can go into the gluconeogenesis pathway. That'll increase uti incidence, shred your kidneys with proteinurea, and lead to ketoacidosis when you're dealing with the other half of the proteins and all the other lipids, which before it kills you will allow you to excrete that waste from you lungs while you smell like you've been huffing nail polish remover. I think the out the butt option is generally preferable."
+lang: "en"
+isAdult: false
+favouriteCount: 0
+score: 0
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date: 2024-01-03 18:39:49.0 +01:00
}
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"@LesserAbe@lemmy.world"
"@gibmiser@lemmy.world"
"@roguetrick@kbin.social"
"@theneverfox@pawb.social"
"@LesserAbe@lemmy.world"
"@gibmiser@lemmy.world"
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"@gibmiser@lemmy.world"
"@roguetrick@kbin.social"
"@theneverfox@pawb.social"
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-bodyTs: "'allow':51 'butt':76 'carbohydr':7 'deal':32 'excret':54 'general':79 'gluconeogenesi':13 'go':10 'half':3,36 'huf':67 'incid':19 'increas':17 'ketoacidosi':28 'kidney':22 'kill':48 'lead':26 'like':63 'lipid':44 'll':16 'lung':59 'nail':68 'option':77 'pathway':14 'polish':69 'prefer':80 'protein':5,39 'proteinurea':24 're':31 'remov':70 'shred':20 'smell':62 'think':72 'uti':18 've':65 'wast':56"
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+editedAt: DateTimeImmutable @1711295080 {#2261
date: 2024-03-24 16:44:40.0 +01:00
}
+createdAt: DateTimeImmutable @1704303589 {#2220
date: 2024-01-03 18:39:49.0 +01:00
}
+"title": 277034
}
2 => App\Entity\EntryComment {#2262
+user: App\Entity\User {#260 …}
+entry: Proxies\__CG__\App\Entity\Entry {#2222 …2}
+magazine: Proxies\__CG__\App\Entity\Magazine {#1550 …}
+image: null
+parent: Proxies\__CG__\App\Entity\EntryComment {#2263 …}
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+body: """
That's a complicated question, because yes there are weight loss drugs that mimic certain aspects of diabetes, but through different mechanisms of action. A good example is a SGLT2 inhibitor medication, which explicitly creates glycosuria in a non-diabetic person, which is kind of a defining feature of diabetes mellitus. While that's a generally very effective drug and should be used more for obesity control, it will also increase incidence of UTI and (due to a loss of sugar and thus forcing metabolism of fats/proteins) ketoacidosis.\n
\n
The insulin/glucagon relationship is why diabetic ketoacidosis only shows up in type 1 diabetics and not type 2 (they get what's called hyperosmolar hyperglycemic state), and is an important part of liver fat production. Ketoacidosis is generally associated with that but is also present in starvation and alcoholics that don't eat actual food.\n
\n
The greater point I was making wasn't about carbohydrates turning into fats. It's that most fats and about half of proteins cannot be turned into carbohydrates. They must be metabolized into ketones and then acetone. If you absorb them and cannot store them like in the OP's hypothetical, you'll end up having to immediately metabolize them and will end up with ketoacidosis.
"""
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+isAdult: false
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+score: 0
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date: 2024-01-04 18:23:02.0 +01:00
}
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"@LesserAbe@lemmy.world"
"@gibmiser@lemmy.world"
"@roguetrick@kbin.social"
"@theneverfox@pawb.social"
"@LesserAbe@lemmy.world"
"@gibmiser@lemmy.world"
"@roguetrick@kbin.social"
"@theneverfox@pawb.social"
"@LesserAbe@lemmy.world"
"@gibmiser@lemmy.world"
"@roguetrick@kbin.social"
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"@gibmiser@lemmy.world"
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+children: Doctrine\ORM\PersistentCollection {#2264 …}
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-id: 280094
-bodyTs: "'1':102 '2':107 'absorb':184 'aceton':181 'action':24 'actual':143 'alcohol':138 'also':71,133 'aspect':16 'associ':128 'call':112 'cannot':168,187 'carbohydr':154,172 'certain':15 'complic':4 'control':68 'creat':35 'defin':48 'diabet':18,41,51,95,103 'differ':21 'drug':12,60 'due':77 'eat':142 'effect':59 'end':198,207 'exampl':27 'explicit':34 'fat':123,157,162 'fats/proteins':88 'featur':49 'food':144 'forc':85 'general':57,127 'get':109 'glycosuria':36 'good':26 'greater':146 'half':165 'hyperglycem':114 'hyperosmolar':113 'hypothet':195 'immedi':202 'import':119 'incid':73 'increas':72 'inhibitor':31 'insulin/glucagon':91 'ketoacidosi':89,96,125,210 'keton':178 'kind':45 'like':190 'liver':122 'll':197 'loss':11,80 'make':150 'mechan':22 'medic':32 'mellitus':52 'metabol':86,176,203 'mimic':14 'must':174 'non':40 'non-diabet':39 'obes':67 'op':193 'part':120 'person':42 'point':147 'present':134 'product':124 'protein':167 'question':5 'relationship':92 'sglt2':30 'show':98 'starvat':136 'state':115 'store':188 'sugar':82 'thus':84 'turn':155,170 'type':101,106 'use':64 'uti':75 'wasn':151 'weight':10 'yes':7"
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date: 2024-04-02 15:53:56.0 +02:00
}
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date: 2024-01-04 18:23:02.0 +01:00
}
+"title": 280094
}
0 => App\Entity\EntryComment {#2208
+user: App\Entity\User {#260 …}
+entry: Proxies\__CG__\App\Entity\Entry {#2222 …2}
+magazine: Proxies\__CG__\App\Entity\Magazine {#1550 …}
+image: null
+parent: Proxies\__CG__\App\Entity\EntryComment {#2209 …}
+root: Proxies\__CG__\App\Entity\EntryComment {#2219 …}
+body: """
It's all about degree. Essentially what I'm talking about is severe dysregulation of lipid/protein metabolism that is generally only seen in type 1 diabetics, but it's the natural extension of extreme lipid and protein metabolism. I'm not familiar with that supplement, however.\n
\n
As far as you watching yourself for it, don't worry, you'd notice being acidotic. I've seen it plenty of times at the hospital and you'd be huffing and puffing the acid off with terrible stomach pain and be generally having a bad day. While that supplement is associated with urine acetone increases, it's not quite the degree that I'm taking about.\n
\n
Essentially, inhibiting fat production and storage and eliminating it result in very different effects. Insulin, by the way, is a very important mediator in fat production and storage. It's why so many new diagnosis type 1 diabetic children are often underweight.
"""
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date: 2024-01-04 08:45:01.0 +01:00
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"@LesserAbe@lemmy.world"
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date: 2024-03-27 03:44:32.0 +01:00
}
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date: 2024-01-04 08:45:01.0 +01:00
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date: 2024-03-21 03:39:54.0 +01:00
}
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date: 2024-01-02 20:40:53.0 +01:00
}
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|
Show voter details
|
| 4 |
DENIED
|
moderate
|
Proxies\__CG__\App\Entity\Entry {#2222
+user: Proxies\__CG__\App\Entity\User {#2873 …}
+magazine: Proxies\__CG__\App\Entity\Magazine {#1550 …}
+image: null
+domain: Proxies\__CG__\App\Entity\Domain {#2875 …}
+slug: "What-are-some-small-things-we-should-change-about-the"
+title: "What are some small things we should change about the human body?"
+url: null
+body: """
Ripping off a post I just saw in the Isaac Arthur subreddit. Imagining we work out the technical ability. Examples they suggested were:\n
\n
- No periods\n
- No balding\n
- No nausea\n
- No body odor\n
- Resistance to obesity and muscle wasting
"""
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1 => App\Entity\EntryComment {#2218
+user: App\Entity\User {#260 …}
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+magazine: Proxies\__CG__\App\Entity\Magazine {#1550 …}
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+root: Proxies\__CG__\App\Entity\EntryComment {#2219 …}
+body: "Only for half of protein and carbohydrates that can go into the gluconeogenesis pathway. That'll increase uti incidence, shred your kidneys with proteinurea, and lead to ketoacidosis when you're dealing with the other half of the proteins and all the other lipids, which before it kills you will allow you to excrete that waste from you lungs while you smell like you've been huffing nail polish remover. I think the out the butt option is generally preferable."
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date: 2024-01-03 18:39:49.0 +01:00
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"@LesserAbe@lemmy.world"
"@gibmiser@lemmy.world"
"@roguetrick@kbin.social"
"@theneverfox@pawb.social"
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-id: 277034
-bodyTs: "'allow':51 'butt':76 'carbohydr':7 'deal':32 'excret':54 'general':79 'gluconeogenesi':13 'go':10 'half':3,36 'huf':67 'incid':19 'increas':17 'ketoacidosi':28 'kidney':22 'kill':48 'lead':26 'like':63 'lipid':44 'll':16 'lung':59 'nail':68 'option':77 'pathway':14 'polish':69 'prefer':80 'protein':5,39 'proteinurea':24 're':31 'remov':70 'shred':20 'smell':62 'think':72 'uti':18 've':65 'wast':56"
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+editedAt: DateTimeImmutable @1711295080 {#2261
date: 2024-03-24 16:44:40.0 +01:00
}
+createdAt: DateTimeImmutable @1704303589 {#2220
date: 2024-01-03 18:39:49.0 +01:00
}
+"title": 277034
}
2 => App\Entity\EntryComment {#2262
+user: App\Entity\User {#260 …}
+entry: Proxies\__CG__\App\Entity\Entry {#2222 …2}
+magazine: Proxies\__CG__\App\Entity\Magazine {#1550 …}
+image: null
+parent: Proxies\__CG__\App\Entity\EntryComment {#2263 …}
+root: Proxies\__CG__\App\Entity\EntryComment {#2219 …}
+body: """
That's a complicated question, because yes there are weight loss drugs that mimic certain aspects of diabetes, but through different mechanisms of action. A good example is a SGLT2 inhibitor medication, which explicitly creates glycosuria in a non-diabetic person, which is kind of a defining feature of diabetes mellitus. While that's a generally very effective drug and should be used more for obesity control, it will also increase incidence of UTI and (due to a loss of sugar and thus forcing metabolism of fats/proteins) ketoacidosis.\n
\n
The insulin/glucagon relationship is why diabetic ketoacidosis only shows up in type 1 diabetics and not type 2 (they get what's called hyperosmolar hyperglycemic state), and is an important part of liver fat production. Ketoacidosis is generally associated with that but is also present in starvation and alcoholics that don't eat actual food.\n
\n
The greater point I was making wasn't about carbohydrates turning into fats. It's that most fats and about half of proteins cannot be turned into carbohydrates. They must be metabolized into ketones and then acetone. If you absorb them and cannot store them like in the OP's hypothetical, you'll end up having to immediately metabolize them and will end up with ketoacidosis.
"""
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date: 2024-01-04 18:23:02.0 +01:00
}
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"@LesserAbe@lemmy.world"
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]
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-id: 280094
-bodyTs: "'1':102 '2':107 'absorb':184 'aceton':181 'action':24 'actual':143 'alcohol':138 'also':71,133 'aspect':16 'associ':128 'call':112 'cannot':168,187 'carbohydr':154,172 'certain':15 'complic':4 'control':68 'creat':35 'defin':48 'diabet':18,41,51,95,103 'differ':21 'drug':12,60 'due':77 'eat':142 'effect':59 'end':198,207 'exampl':27 'explicit':34 'fat':123,157,162 'fats/proteins':88 'featur':49 'food':144 'forc':85 'general':57,127 'get':109 'glycosuria':36 'good':26 'greater':146 'half':165 'hyperglycem':114 'hyperosmolar':113 'hypothet':195 'immedi':202 'import':119 'incid':73 'increas':72 'inhibitor':31 'insulin/glucagon':91 'ketoacidosi':89,96,125,210 'keton':178 'kind':45 'like':190 'liver':122 'll':197 'loss':11,80 'make':150 'mechan':22 'medic':32 'mellitus':52 'metabol':86,176,203 'mimic':14 'must':174 'non':40 'non-diabet':39 'obes':67 'op':193 'part':120 'person':42 'point':147 'present':134 'product':124 'protein':167 'question':5 'relationship':92 'sglt2':30 'show':98 'starvat':136 'state':115 'store':188 'sugar':82 'thus':84 'turn':155,170 'type':101,106 'use':64 'uti':75 'wasn':151 'weight':10 'yes':7"
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+editedAt: DateTimeImmutable @1712066036 {#2253
date: 2024-04-02 15:53:56.0 +02:00
}
+createdAt: DateTimeImmutable @1704388982 {#2265
date: 2024-01-04 18:23:02.0 +01:00
}
+"title": 280094
}
0 => App\Entity\EntryComment {#2208
+user: App\Entity\User {#260 …}
+entry: Proxies\__CG__\App\Entity\Entry {#2222 …2}
+magazine: Proxies\__CG__\App\Entity\Magazine {#1550 …}
+image: null
+parent: Proxies\__CG__\App\Entity\EntryComment {#2209 …}
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+body: """
It's all about degree. Essentially what I'm talking about is severe dysregulation of lipid/protein metabolism that is generally only seen in type 1 diabetics, but it's the natural extension of extreme lipid and protein metabolism. I'm not familiar with that supplement, however.\n
\n
As far as you watching yourself for it, don't worry, you'd notice being acidotic. I've seen it plenty of times at the hospital and you'd be huffing and puffing the acid off with terrible stomach pain and be generally having a bad day. While that supplement is associated with urine acetone increases, it's not quite the degree that I'm taking about.\n
\n
Essentially, inhibiting fat production and storage and eliminating it result in very different effects. Insulin, by the way, is a very important mediator in fat production and storage. It's why so many new diagnosis type 1 diabetic children are often underweight.
"""
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date: 2024-01-04 08:45:01.0 +01:00
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"@LesserAbe@lemmy.world"
"@gibmiser@lemmy.world"
"@roguetrick@kbin.social"
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date: 2024-03-27 03:44:32.0 +01:00
}
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date: 2024-01-04 08:45:01.0 +01:00
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]
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date: 2024-03-21 03:39:54.0 +01:00
}
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date: 2024-01-02 20:40:53.0 +01:00
}
+__isInitialized__: true
…2
} |
|
Show voter details
|
| 5 |
DENIED
|
ROLE_USER
|
null |
|
Show voter details
|
| 6 |
DENIED
|
moderate
|
App\Entity\EntryComment {#2218
+user: App\Entity\User {#260 …}
+entry: Proxies\__CG__\App\Entity\Entry {#2222
+user: Proxies\__CG__\App\Entity\User {#2873 …}
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+domain: Proxies\__CG__\App\Entity\Domain {#2875 …}
+slug: "What-are-some-small-things-we-should-change-about-the"
+title: "What are some small things we should change about the human body?"
+url: null
+body: """
Ripping off a post I just saw in the Isaac Arthur subreddit. Imagining we work out the technical ability. Examples they suggested were:\n
\n
- No periods\n
- No balding\n
- No nausea\n
- No body odor\n
- Resistance to obesity and muscle wasting
"""
+type: "article"
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1 => App\Entity\EntryComment {#2218}
2 => App\Entity\EntryComment {#2262
+user: App\Entity\User {#260 …}
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+magazine: Proxies\__CG__\App\Entity\Magazine {#1550 …}
+image: null
+parent: Proxies\__CG__\App\Entity\EntryComment {#2263 …}
+root: Proxies\__CG__\App\Entity\EntryComment {#2219 …}
+body: """
That's a complicated question, because yes there are weight loss drugs that mimic certain aspects of diabetes, but through different mechanisms of action. A good example is a SGLT2 inhibitor medication, which explicitly creates glycosuria in a non-diabetic person, which is kind of a defining feature of diabetes mellitus. While that's a generally very effective drug and should be used more for obesity control, it will also increase incidence of UTI and (due to a loss of sugar and thus forcing metabolism of fats/proteins) ketoacidosis.\n
\n
The insulin/glucagon relationship is why diabetic ketoacidosis only shows up in type 1 diabetics and not type 2 (they get what's called hyperosmolar hyperglycemic state), and is an important part of liver fat production. Ketoacidosis is generally associated with that but is also present in starvation and alcoholics that don't eat actual food.\n
\n
The greater point I was making wasn't about carbohydrates turning into fats. It's that most fats and about half of proteins cannot be turned into carbohydrates. They must be metabolized into ketones and then acetone. If you absorb them and cannot store them like in the OP's hypothetical, you'll end up having to immediately metabolize them and will end up with ketoacidosis.
"""
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date: 2024-01-04 18:23:02.0 +01:00
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-id: 280094
-bodyTs: "'1':102 '2':107 'absorb':184 'aceton':181 'action':24 'actual':143 'alcohol':138 'also':71,133 'aspect':16 'associ':128 'call':112 'cannot':168,187 'carbohydr':154,172 'certain':15 'complic':4 'control':68 'creat':35 'defin':48 'diabet':18,41,51,95,103 'differ':21 'drug':12,60 'due':77 'eat':142 'effect':59 'end':198,207 'exampl':27 'explicit':34 'fat':123,157,162 'fats/proteins':88 'featur':49 'food':144 'forc':85 'general':57,127 'get':109 'glycosuria':36 'good':26 'greater':146 'half':165 'hyperglycem':114 'hyperosmolar':113 'hypothet':195 'immedi':202 'import':119 'incid':73 'increas':72 'inhibitor':31 'insulin/glucagon':91 'ketoacidosi':89,96,125,210 'keton':178 'kind':45 'like':190 'liver':122 'll':197 'loss':11,80 'make':150 'mechan':22 'medic':32 'mellitus':52 'metabol':86,176,203 'mimic':14 'must':174 'non':40 'non-diabet':39 'obes':67 'op':193 'part':120 'person':42 'point':147 'present':134 'product':124 'protein':167 'question':5 'relationship':92 'sglt2':30 'show':98 'starvat':136 'state':115 'store':188 'sugar':82 'thus':84 'turn':155,170 'type':101,106 'use':64 'uti':75 'wasn':151 'weight':10 'yes':7"
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date: 2024-04-02 15:53:56.0 +02:00
}
+createdAt: DateTimeImmutable @1704388982 {#2265
date: 2024-01-04 18:23:02.0 +01:00
}
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}
0 => App\Entity\EntryComment {#2208
+user: App\Entity\User {#260 …}
+entry: Proxies\__CG__\App\Entity\Entry {#2222 …2}
+magazine: Proxies\__CG__\App\Entity\Magazine {#1550 …}
+image: null
+parent: Proxies\__CG__\App\Entity\EntryComment {#2209 …}
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+body: """
It's all about degree. Essentially what I'm talking about is severe dysregulation of lipid/protein metabolism that is generally only seen in type 1 diabetics, but it's the natural extension of extreme lipid and protein metabolism. I'm not familiar with that supplement, however.\n
\n
As far as you watching yourself for it, don't worry, you'd notice being acidotic. I've seen it plenty of times at the hospital and you'd be huffing and puffing the acid off with terrible stomach pain and be generally having a bad day. While that supplement is associated with urine acetone increases, it's not quite the degree that I'm taking about.\n
\n
Essentially, inhibiting fat production and storage and eliminating it result in very different effects. Insulin, by the way, is a very important mediator in fat production and storage. It's why so many new diagnosis type 1 diabetic children are often underweight.
"""
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+body: "Only for half of protein and carbohydrates that can go into the gluconeogenesis pathway. That'll increase uti incidence, shred your kidneys with proteinurea, and lead to ketoacidosis when you're dealing with the other half of the proteins and all the other lipids, which before it kills you will allow you to excrete that waste from you lungs while you smell like you've been huffing nail polish remover. I think the out the butt option is generally preferable."
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}
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} |
|
Show voter details
|
| 7 |
DENIED
|
edit
|
App\Entity\EntryComment {#2218
+user: App\Entity\User {#260 …}
+entry: Proxies\__CG__\App\Entity\Entry {#2222
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+slug: "What-are-some-small-things-we-should-change-about-the"
+title: "What are some small things we should change about the human body?"
+url: null
+body: """
Ripping off a post I just saw in the Isaac Arthur subreddit. Imagining we work out the technical ability. Examples they suggested were:\n
\n
- No periods\n
- No balding\n
- No nausea\n
- No body odor\n
- Resistance to obesity and muscle wasting
"""
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1 => App\Entity\EntryComment {#2218}
2 => App\Entity\EntryComment {#2262
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+body: """
That's a complicated question, because yes there are weight loss drugs that mimic certain aspects of diabetes, but through different mechanisms of action. A good example is a SGLT2 inhibitor medication, which explicitly creates glycosuria in a non-diabetic person, which is kind of a defining feature of diabetes mellitus. While that's a generally very effective drug and should be used more for obesity control, it will also increase incidence of UTI and (due to a loss of sugar and thus forcing metabolism of fats/proteins) ketoacidosis.\n
\n
The insulin/glucagon relationship is why diabetic ketoacidosis only shows up in type 1 diabetics and not type 2 (they get what's called hyperosmolar hyperglycemic state), and is an important part of liver fat production. Ketoacidosis is generally associated with that but is also present in starvation and alcoholics that don't eat actual food.\n
\n
The greater point I was making wasn't about carbohydrates turning into fats. It's that most fats and about half of proteins cannot be turned into carbohydrates. They must be metabolized into ketones and then acetone. If you absorb them and cannot store them like in the OP's hypothetical, you'll end up having to immediately metabolize them and will end up with ketoacidosis.
"""
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0 => App\Entity\EntryComment {#2208
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+body: """
It's all about degree. Essentially what I'm talking about is severe dysregulation of lipid/protein metabolism that is generally only seen in type 1 diabetics, but it's the natural extension of extreme lipid and protein metabolism. I'm not familiar with that supplement, however.\n
\n
As far as you watching yourself for it, don't worry, you'd notice being acidotic. I've seen it plenty of times at the hospital and you'd be huffing and puffing the acid off with terrible stomach pain and be generally having a bad day. While that supplement is associated with urine acetone increases, it's not quite the degree that I'm taking about.\n
\n
Essentially, inhibiting fat production and storage and eliminating it result in very different effects. Insulin, by the way, is a very important mediator in fat production and storage. It's why so many new diagnosis type 1 diabetic children are often underweight.
"""
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date: 2024-01-02 20:40:53.0 +01:00
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…2
}
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+body: "Only for half of protein and carbohydrates that can go into the gluconeogenesis pathway. That'll increase uti incidence, shred your kidneys with proteinurea, and lead to ketoacidosis when you're dealing with the other half of the proteins and all the other lipids, which before it kills you will allow you to excrete that waste from you lungs while you smell like you've been huffing nail polish remover. I think the out the butt option is generally preferable."
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}
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date: 2024-01-03 18:39:49.0 +01:00
}
+"title": 277034
} |
|
Show voter details
|
| 8 |
DENIED
|
moderate
|
App\Entity\EntryComment {#2218
+user: App\Entity\User {#260 …}
+entry: Proxies\__CG__\App\Entity\Entry {#2222
+user: Proxies\__CG__\App\Entity\User {#2873 …}
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+image: null
+domain: Proxies\__CG__\App\Entity\Domain {#2875 …}
+slug: "What-are-some-small-things-we-should-change-about-the"
+title: "What are some small things we should change about the human body?"
+url: null
+body: """
Ripping off a post I just saw in the Isaac Arthur subreddit. Imagining we work out the technical ability. Examples they suggested were:\n
\n
- No periods\n
- No balding\n
- No nausea\n
- No body odor\n
- Resistance to obesity and muscle wasting
"""
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date: 2024-10-11 21:35:30.0 +02:00
}
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1 => App\Entity\EntryComment {#2218}
2 => App\Entity\EntryComment {#2262
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+image: null
+parent: Proxies\__CG__\App\Entity\EntryComment {#2263 …}
+root: Proxies\__CG__\App\Entity\EntryComment {#2219 …}
+body: """
That's a complicated question, because yes there are weight loss drugs that mimic certain aspects of diabetes, but through different mechanisms of action. A good example is a SGLT2 inhibitor medication, which explicitly creates glycosuria in a non-diabetic person, which is kind of a defining feature of diabetes mellitus. While that's a generally very effective drug and should be used more for obesity control, it will also increase incidence of UTI and (due to a loss of sugar and thus forcing metabolism of fats/proteins) ketoacidosis.\n
\n
The insulin/glucagon relationship is why diabetic ketoacidosis only shows up in type 1 diabetics and not type 2 (they get what's called hyperosmolar hyperglycemic state), and is an important part of liver fat production. Ketoacidosis is generally associated with that but is also present in starvation and alcoholics that don't eat actual food.\n
\n
The greater point I was making wasn't about carbohydrates turning into fats. It's that most fats and about half of proteins cannot be turned into carbohydrates. They must be metabolized into ketones and then acetone. If you absorb them and cannot store them like in the OP's hypothetical, you'll end up having to immediately metabolize them and will end up with ketoacidosis.
"""
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date: 2024-01-04 18:23:02.0 +01:00
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"@gibmiser@lemmy.world"
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date: 2024-04-02 15:53:56.0 +02:00
}
+createdAt: DateTimeImmutable @1704388982 {#2265
date: 2024-01-04 18:23:02.0 +01:00
}
+"title": 280094
}
0 => App\Entity\EntryComment {#2208
+user: App\Entity\User {#260 …}
+entry: Proxies\__CG__\App\Entity\Entry {#2222 …2}
+magazine: Proxies\__CG__\App\Entity\Magazine {#1550 …}
+image: null
+parent: Proxies\__CG__\App\Entity\EntryComment {#2209 …}
+root: Proxies\__CG__\App\Entity\EntryComment {#2219 …}
+body: """
It's all about degree. Essentially what I'm talking about is severe dysregulation of lipid/protein metabolism that is generally only seen in type 1 diabetics, but it's the natural extension of extreme lipid and protein metabolism. I'm not familiar with that supplement, however.\n
\n
As far as you watching yourself for it, don't worry, you'd notice being acidotic. I've seen it plenty of times at the hospital and you'd be huffing and puffing the acid off with terrible stomach pain and be generally having a bad day. While that supplement is associated with urine acetone increases, it's not quite the degree that I'm taking about.\n
\n
Essentially, inhibiting fat production and storage and eliminating it result in very different effects. Insulin, by the way, is a very important mediator in fat production and storage. It's why so many new diagnosis type 1 diabetic children are often underweight.
"""
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date: 2024-01-04 08:45:01.0 +01:00
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-id: 278949
-bodyTs: "'1':25,150 'aceton':101 'acid':81 'acidot':62 'associ':98 'bad':92 'children':152 'd':59,75 'day':93 'degre':5,108 'diabet':26,151 'diagnosi':148 'differ':126 'dysregul':14 'effect':127 'elimin':121 'essenti':6,114 'extens':32 'extrem':34 'familiar':42 'far':48 'fat':116,138 'general':20,89 'hospit':72 'howev':46 'huf':77 'import':135 'increas':102 'inhibit':115 'insulin':128 'lipid':35 'lipid/protein':16 'm':9,40,111 'mani':146 'mediat':136 'metabol':17,38 'natur':31 'new':147 'notic':60 'often':154 'pain':86 'plenti':67 'product':117,139 'protein':37 'puf':79 'quit':106 'result':123 'seen':22,65 'sever':13 'stomach':85 'storag':119,141 'supplement':45,96 'take':112 'talk':10 'terribl':84 'time':69 'type':24,149 'underweight':155 'urin':100 've':64 'watch':51 'way':131 'worri':57"
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date: 2024-01-02 20:40:53.0 +01:00
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…2
}
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+body: "Only for half of protein and carbohydrates that can go into the gluconeogenesis pathway. That'll increase uti incidence, shred your kidneys with proteinurea, and lead to ketoacidosis when you're dealing with the other half of the proteins and all the other lipids, which before it kills you will allow you to excrete that waste from you lungs while you smell like you've been huffing nail polish remover. I think the out the butt option is generally preferable."
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date: 2024-01-03 18:39:49.0 +01:00
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date: 2024-01-03 18:39:49.0 +01:00
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+"title": 277034
} |
|
Show voter details
|
| 9 |
DENIED
|
ROLE_USER
|
null |
|
Show voter details
|
| 10 |
DENIED
|
moderate
|
App\Entity\EntryComment {#2262
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Ripping off a post I just saw in the Isaac Arthur subreddit. Imagining we work out the technical ability. Examples they suggested were:\n
\n
- No periods\n
- No balding\n
- No nausea\n
- No body odor\n
- Resistance to obesity and muscle wasting
"""
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1 => App\Entity\EntryComment {#2218
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+body: "Only for half of protein and carbohydrates that can go into the gluconeogenesis pathway. That'll increase uti incidence, shred your kidneys with proteinurea, and lead to ketoacidosis when you're dealing with the other half of the proteins and all the other lipids, which before it kills you will allow you to excrete that waste from you lungs while you smell like you've been huffing nail polish remover. I think the out the butt option is generally preferable."
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}
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date: 2024-01-03 18:39:49.0 +01:00
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}
2 => App\Entity\EntryComment {#2262}
0 => App\Entity\EntryComment {#2208
+user: App\Entity\User {#260 …}
+entry: Proxies\__CG__\App\Entity\Entry {#2222 …2}
+magazine: Proxies\__CG__\App\Entity\Magazine {#1550 …}
+image: null
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+body: """
It's all about degree. Essentially what I'm talking about is severe dysregulation of lipid/protein metabolism that is generally only seen in type 1 diabetics, but it's the natural extension of extreme lipid and protein metabolism. I'm not familiar with that supplement, however.\n
\n
As far as you watching yourself for it, don't worry, you'd notice being acidotic. I've seen it plenty of times at the hospital and you'd be huffing and puffing the acid off with terrible stomach pain and be generally having a bad day. While that supplement is associated with urine acetone increases, it's not quite the degree that I'm taking about.\n
\n
Essentially, inhibiting fat production and storage and eliminating it result in very different effects. Insulin, by the way, is a very important mediator in fat production and storage. It's why so many new diagnosis type 1 diabetic children are often underweight.
"""
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date: 2024-03-21 03:39:54.0 +01:00
}
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date: 2024-01-02 20:40:53.0 +01:00
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…2
}
+magazine: Proxies\__CG__\App\Entity\Magazine {#1550 …}
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+body: """
That's a complicated question, because yes there are weight loss drugs that mimic certain aspects of diabetes, but through different mechanisms of action. A good example is a SGLT2 inhibitor medication, which explicitly creates glycosuria in a non-diabetic person, which is kind of a defining feature of diabetes mellitus. While that's a generally very effective drug and should be used more for obesity control, it will also increase incidence of UTI and (due to a loss of sugar and thus forcing metabolism of fats/proteins) ketoacidosis.\n
\n
The insulin/glucagon relationship is why diabetic ketoacidosis only shows up in type 1 diabetics and not type 2 (they get what's called hyperosmolar hyperglycemic state), and is an important part of liver fat production. Ketoacidosis is generally associated with that but is also present in starvation and alcoholics that don't eat actual food.\n
\n
The greater point I was making wasn't about carbohydrates turning into fats. It's that most fats and about half of proteins cannot be turned into carbohydrates. They must be metabolized into ketones and then acetone. If you absorb them and cannot store them like in the OP's hypothetical, you'll end up having to immediately metabolize them and will end up with ketoacidosis.
"""
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date: 2024-04-02 15:53:56.0 +02:00
}
+createdAt: DateTimeImmutable @1704388982 {#2265
date: 2024-01-04 18:23:02.0 +01:00
}
+"title": 280094
} |
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Show voter details
|
| 11 |
DENIED
|
edit
|
App\Entity\EntryComment {#2262
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+title: "What are some small things we should change about the human body?"
+url: null
+body: """
Ripping off a post I just saw in the Isaac Arthur subreddit. Imagining we work out the technical ability. Examples they suggested were:\n
\n
- No periods\n
- No balding\n
- No nausea\n
- No body odor\n
- Resistance to obesity and muscle wasting
"""
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1 => App\Entity\EntryComment {#2218
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+body: "Only for half of protein and carbohydrates that can go into the gluconeogenesis pathway. That'll increase uti incidence, shred your kidneys with proteinurea, and lead to ketoacidosis when you're dealing with the other half of the proteins and all the other lipids, which before it kills you will allow you to excrete that waste from you lungs while you smell like you've been huffing nail polish remover. I think the out the butt option is generally preferable."
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date: 2024-01-03 18:39:49.0 +01:00
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date: 2024-03-24 16:44:40.0 +01:00
}
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date: 2024-01-03 18:39:49.0 +01:00
}
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}
2 => App\Entity\EntryComment {#2262}
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It's all about degree. Essentially what I'm talking about is severe dysregulation of lipid/protein metabolism that is generally only seen in type 1 diabetics, but it's the natural extension of extreme lipid and protein metabolism. I'm not familiar with that supplement, however.\n
\n
As far as you watching yourself for it, don't worry, you'd notice being acidotic. I've seen it plenty of times at the hospital and you'd be huffing and puffing the acid off with terrible stomach pain and be generally having a bad day. While that supplement is associated with urine acetone increases, it's not quite the degree that I'm taking about.\n
\n
Essentially, inhibiting fat production and storage and eliminating it result in very different effects. Insulin, by the way, is a very important mediator in fat production and storage. It's why so many new diagnosis type 1 diabetic children are often underweight.
"""
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date: 2024-03-21 03:39:54.0 +01:00
}
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date: 2024-01-02 20:40:53.0 +01:00
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…2
}
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+body: """
That's a complicated question, because yes there are weight loss drugs that mimic certain aspects of diabetes, but through different mechanisms of action. A good example is a SGLT2 inhibitor medication, which explicitly creates glycosuria in a non-diabetic person, which is kind of a defining feature of diabetes mellitus. While that's a generally very effective drug and should be used more for obesity control, it will also increase incidence of UTI and (due to a loss of sugar and thus forcing metabolism of fats/proteins) ketoacidosis.\n
\n
The insulin/glucagon relationship is why diabetic ketoacidosis only shows up in type 1 diabetics and not type 2 (they get what's called hyperosmolar hyperglycemic state), and is an important part of liver fat production. Ketoacidosis is generally associated with that but is also present in starvation and alcoholics that don't eat actual food.\n
\n
The greater point I was making wasn't about carbohydrates turning into fats. It's that most fats and about half of proteins cannot be turned into carbohydrates. They must be metabolized into ketones and then acetone. If you absorb them and cannot store them like in the OP's hypothetical, you'll end up having to immediately metabolize them and will end up with ketoacidosis.
"""
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}
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date: 2024-01-04 18:23:02.0 +01:00
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+"title": 280094
} |
|
Show voter details
|
| 12 |
DENIED
|
moderate
|
App\Entity\EntryComment {#2262
+user: App\Entity\User {#260 …}
+entry: Proxies\__CG__\App\Entity\Entry {#2222
+user: Proxies\__CG__\App\Entity\User {#2873 …}
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+domain: Proxies\__CG__\App\Entity\Domain {#2875 …}
+slug: "What-are-some-small-things-we-should-change-about-the"
+title: "What are some small things we should change about the human body?"
+url: null
+body: """
Ripping off a post I just saw in the Isaac Arthur subreddit. Imagining we work out the technical ability. Examples they suggested were:\n
\n
- No periods\n
- No balding\n
- No nausea\n
- No body odor\n
- Resistance to obesity and muscle wasting
"""
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1 => App\Entity\EntryComment {#2218
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+body: "Only for half of protein and carbohydrates that can go into the gluconeogenesis pathway. That'll increase uti incidence, shred your kidneys with proteinurea, and lead to ketoacidosis when you're dealing with the other half of the proteins and all the other lipids, which before it kills you will allow you to excrete that waste from you lungs while you smell like you've been huffing nail polish remover. I think the out the butt option is generally preferable."
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date: 2024-01-03 18:39:49.0 +01:00
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date: 2024-03-24 16:44:40.0 +01:00
}
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date: 2024-01-03 18:39:49.0 +01:00
}
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}
2 => App\Entity\EntryComment {#2262}
0 => App\Entity\EntryComment {#2208
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+body: """
It's all about degree. Essentially what I'm talking about is severe dysregulation of lipid/protein metabolism that is generally only seen in type 1 diabetics, but it's the natural extension of extreme lipid and protein metabolism. I'm not familiar with that supplement, however.\n
\n
As far as you watching yourself for it, don't worry, you'd notice being acidotic. I've seen it plenty of times at the hospital and you'd be huffing and puffing the acid off with terrible stomach pain and be generally having a bad day. While that supplement is associated with urine acetone increases, it's not quite the degree that I'm taking about.\n
\n
Essentially, inhibiting fat production and storage and eliminating it result in very different effects. Insulin, by the way, is a very important mediator in fat production and storage. It's why so many new diagnosis type 1 diabetic children are often underweight.
"""
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}
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date: 2024-01-02 20:40:53.0 +01:00
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…2
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That's a complicated question, because yes there are weight loss drugs that mimic certain aspects of diabetes, but through different mechanisms of action. A good example is a SGLT2 inhibitor medication, which explicitly creates glycosuria in a non-diabetic person, which is kind of a defining feature of diabetes mellitus. While that's a generally very effective drug and should be used more for obesity control, it will also increase incidence of UTI and (due to a loss of sugar and thus forcing metabolism of fats/proteins) ketoacidosis.\n
\n
The insulin/glucagon relationship is why diabetic ketoacidosis only shows up in type 1 diabetics and not type 2 (they get what's called hyperosmolar hyperglycemic state), and is an important part of liver fat production. Ketoacidosis is generally associated with that but is also present in starvation and alcoholics that don't eat actual food.\n
\n
The greater point I was making wasn't about carbohydrates turning into fats. It's that most fats and about half of proteins cannot be turned into carbohydrates. They must be metabolized into ketones and then acetone. If you absorb them and cannot store them like in the OP's hypothetical, you'll end up having to immediately metabolize them and will end up with ketoacidosis.
"""
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date: 2024-01-04 18:23:02.0 +01:00
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} |
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Show voter details
|
| 13 |
DENIED
|
ROLE_USER
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null |
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Show voter details
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| 14 |
DENIED
|
moderate
|
App\Entity\EntryComment {#2208
+user: App\Entity\User {#260 …}
+entry: Proxies\__CG__\App\Entity\Entry {#2222
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+title: "What are some small things we should change about the human body?"
+url: null
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Ripping off a post I just saw in the Isaac Arthur subreddit. Imagining we work out the technical ability. Examples they suggested were:\n
\n
- No periods\n
- No balding\n
- No nausea\n
- No body odor\n
- Resistance to obesity and muscle wasting
"""
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1 => App\Entity\EntryComment {#2218
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+entry: Proxies\__CG__\App\Entity\Entry {#2222 …2}
+magazine: Proxies\__CG__\App\Entity\Magazine {#1550 …}
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+body: "Only for half of protein and carbohydrates that can go into the gluconeogenesis pathway. That'll increase uti incidence, shred your kidneys with proteinurea, and lead to ketoacidosis when you're dealing with the other half of the proteins and all the other lipids, which before it kills you will allow you to excrete that waste from you lungs while you smell like you've been huffing nail polish remover. I think the out the butt option is generally preferable."
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}
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date: 2024-01-03 18:39:49.0 +01:00
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}
2 => App\Entity\EntryComment {#2262
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+body: """
That's a complicated question, because yes there are weight loss drugs that mimic certain aspects of diabetes, but through different mechanisms of action. A good example is a SGLT2 inhibitor medication, which explicitly creates glycosuria in a non-diabetic person, which is kind of a defining feature of diabetes mellitus. While that's a generally very effective drug and should be used more for obesity control, it will also increase incidence of UTI and (due to a loss of sugar and thus forcing metabolism of fats/proteins) ketoacidosis.\n
\n
The insulin/glucagon relationship is why diabetic ketoacidosis only shows up in type 1 diabetics and not type 2 (they get what's called hyperosmolar hyperglycemic state), and is an important part of liver fat production. Ketoacidosis is generally associated with that but is also present in starvation and alcoholics that don't eat actual food.\n
\n
The greater point I was making wasn't about carbohydrates turning into fats. It's that most fats and about half of proteins cannot be turned into carbohydrates. They must be metabolized into ketones and then acetone. If you absorb them and cannot store them like in the OP's hypothetical, you'll end up having to immediately metabolize them and will end up with ketoacidosis.
"""
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]
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date: 2024-01-02 20:40:53.0 +01:00
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…2
}
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+body: """
It's all about degree. Essentially what I'm talking about is severe dysregulation of lipid/protein metabolism that is generally only seen in type 1 diabetics, but it's the natural extension of extreme lipid and protein metabolism. I'm not familiar with that supplement, however.\n
\n
As far as you watching yourself for it, don't worry, you'd notice being acidotic. I've seen it plenty of times at the hospital and you'd be huffing and puffing the acid off with terrible stomach pain and be generally having a bad day. While that supplement is associated with urine acetone increases, it's not quite the degree that I'm taking about.\n
\n
Essentially, inhibiting fat production and storage and eliminating it result in very different effects. Insulin, by the way, is a very important mediator in fat production and storage. It's why so many new diagnosis type 1 diabetic children are often underweight.
"""
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}
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date: 2024-01-04 08:45:01.0 +01:00
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} |
|
Show voter details
|
| 15 |
DENIED
|
edit
|
App\Entity\EntryComment {#2208
+user: App\Entity\User {#260 …}
+entry: Proxies\__CG__\App\Entity\Entry {#2222
+user: Proxies\__CG__\App\Entity\User {#2873 …}
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+slug: "What-are-some-small-things-we-should-change-about-the"
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+url: null
+body: """
Ripping off a post I just saw in the Isaac Arthur subreddit. Imagining we work out the technical ability. Examples they suggested were:\n
\n
- No periods\n
- No balding\n
- No nausea\n
- No body odor\n
- Resistance to obesity and muscle wasting
"""
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1 => App\Entity\EntryComment {#2218
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+body: "Only for half of protein and carbohydrates that can go into the gluconeogenesis pathway. That'll increase uti incidence, shred your kidneys with proteinurea, and lead to ketoacidosis when you're dealing with the other half of the proteins and all the other lipids, which before it kills you will allow you to excrete that waste from you lungs while you smell like you've been huffing nail polish remover. I think the out the butt option is generally preferable."
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date: 2024-01-03 18:39:49.0 +01:00
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date: 2024-03-24 16:44:40.0 +01:00
}
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date: 2024-01-03 18:39:49.0 +01:00
}
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}
2 => App\Entity\EntryComment {#2262
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+entry: Proxies\__CG__\App\Entity\Entry {#2222 …2}
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+body: """
That's a complicated question, because yes there are weight loss drugs that mimic certain aspects of diabetes, but through different mechanisms of action. A good example is a SGLT2 inhibitor medication, which explicitly creates glycosuria in a non-diabetic person, which is kind of a defining feature of diabetes mellitus. While that's a generally very effective drug and should be used more for obesity control, it will also increase incidence of UTI and (due to a loss of sugar and thus forcing metabolism of fats/proteins) ketoacidosis.\n
\n
The insulin/glucagon relationship is why diabetic ketoacidosis only shows up in type 1 diabetics and not type 2 (they get what's called hyperosmolar hyperglycemic state), and is an important part of liver fat production. Ketoacidosis is generally associated with that but is also present in starvation and alcoholics that don't eat actual food.\n
\n
The greater point I was making wasn't about carbohydrates turning into fats. It's that most fats and about half of proteins cannot be turned into carbohydrates. They must be metabolized into ketones and then acetone. If you absorb them and cannot store them like in the OP's hypothetical, you'll end up having to immediately metabolize them and will end up with ketoacidosis.
"""
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date: 2024-01-02 20:40:53.0 +01:00
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}
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+body: """
It's all about degree. Essentially what I'm talking about is severe dysregulation of lipid/protein metabolism that is generally only seen in type 1 diabetics, but it's the natural extension of extreme lipid and protein metabolism. I'm not familiar with that supplement, however.\n
\n
As far as you watching yourself for it, don't worry, you'd notice being acidotic. I've seen it plenty of times at the hospital and you'd be huffing and puffing the acid off with terrible stomach pain and be generally having a bad day. While that supplement is associated with urine acetone increases, it's not quite the degree that I'm taking about.\n
\n
Essentially, inhibiting fat production and storage and eliminating it result in very different effects. Insulin, by the way, is a very important mediator in fat production and storage. It's why so many new diagnosis type 1 diabetic children are often underweight.
"""
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Show voter details
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| 16 |
DENIED
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moderate
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App\Entity\EntryComment {#2208
+user: App\Entity\User {#260 …}
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+user: Proxies\__CG__\App\Entity\User {#2873 …}
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+slug: "What-are-some-small-things-we-should-change-about-the"
+title: "What are some small things we should change about the human body?"
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+body: """
Ripping off a post I just saw in the Isaac Arthur subreddit. Imagining we work out the technical ability. Examples they suggested were:\n
\n
- No periods\n
- No balding\n
- No nausea\n
- No body odor\n
- Resistance to obesity and muscle wasting
"""
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1 => App\Entity\EntryComment {#2218
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+body: "Only for half of protein and carbohydrates that can go into the gluconeogenesis pathway. That'll increase uti incidence, shred your kidneys with proteinurea, and lead to ketoacidosis when you're dealing with the other half of the proteins and all the other lipids, which before it kills you will allow you to excrete that waste from you lungs while you smell like you've been huffing nail polish remover. I think the out the butt option is generally preferable."
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}
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date: 2024-01-03 18:39:49.0 +01:00
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2 => App\Entity\EntryComment {#2262
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+body: """
That's a complicated question, because yes there are weight loss drugs that mimic certain aspects of diabetes, but through different mechanisms of action. A good example is a SGLT2 inhibitor medication, which explicitly creates glycosuria in a non-diabetic person, which is kind of a defining feature of diabetes mellitus. While that's a generally very effective drug and should be used more for obesity control, it will also increase incidence of UTI and (due to a loss of sugar and thus forcing metabolism of fats/proteins) ketoacidosis.\n
\n
The insulin/glucagon relationship is why diabetic ketoacidosis only shows up in type 1 diabetics and not type 2 (they get what's called hyperosmolar hyperglycemic state), and is an important part of liver fat production. Ketoacidosis is generally associated with that but is also present in starvation and alcoholics that don't eat actual food.\n
\n
The greater point I was making wasn't about carbohydrates turning into fats. It's that most fats and about half of proteins cannot be turned into carbohydrates. They must be metabolized into ketones and then acetone. If you absorb them and cannot store them like in the OP's hypothetical, you'll end up having to immediately metabolize them and will end up with ketoacidosis.
"""
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+body: """
It's all about degree. Essentially what I'm talking about is severe dysregulation of lipid/protein metabolism that is generally only seen in type 1 diabetics, but it's the natural extension of extreme lipid and protein metabolism. I'm not familiar with that supplement, however.\n
\n
As far as you watching yourself for it, don't worry, you'd notice being acidotic. I've seen it plenty of times at the hospital and you'd be huffing and puffing the acid off with terrible stomach pain and be generally having a bad day. While that supplement is associated with urine acetone increases, it's not quite the degree that I'm taking about.\n
\n
Essentially, inhibiting fat production and storage and eliminating it result in very different effects. Insulin, by the way, is a very important mediator in fat production and storage. It's why so many new diagnosis type 1 diabetic children are often underweight.
"""
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date: 2024-01-04 08:45:01.0 +01:00
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} |
|
Show voter details
|
| 17 |
DENIED
|
ROLE_USER
|
null |
|
Show voter details
|
| 18 |
DENIED
|
moderate
|
Proxies\__CG__\App\Entity\Entry {#2213
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+slug: "friendly-PSA-not-all-jews-are-white"
+title: "friendly PSA: not all jews are white"
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+body: "That's a right winger position on the one state solution. I'm not implying that's the only position, but that's essentially the path Israel is walking down right now. The left wing one state solution is much less problematic."
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|
Show voter details
|
| 19 |
DENIED
|
edit
|
Proxies\__CG__\App\Entity\Entry {#2213
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…2
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|
Show voter details
|
| 20 |
DENIED
|
moderate
|
Proxies\__CG__\App\Entity\Entry {#2213
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…2
} |
|
Show voter details
|
| 21 |
DENIED
|
ROLE_USER
|
null |
|
Show voter details
|
| 22 |
DENIED
|
moderate
|
App\Entity\EntryComment {#2214
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|
Show voter details
|
| 23 |
DENIED
|
edit
|
App\Entity\EntryComment {#2214
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|
Show voter details
|
| 24 |
DENIED
|
moderate
|
App\Entity\EntryComment {#2214
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|
Show voter details
|
| 25 |
DENIED
|
ROLE_USER
|
null |
|
Show voter details
|
| 26 |
DENIED
|
moderate
|
Proxies\__CG__\App\Entity\Entry {#2103
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Show voter details
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| 27 |
DENIED
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edit
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Proxies\__CG__\App\Entity\Entry {#2103
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Show voter details
|
| 28 |
DENIED
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moderate
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Proxies\__CG__\App\Entity\Entry {#2103
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|
Show voter details
|
| 29 |
DENIED
|
ROLE_USER
|
null |
|
Show voter details
|
| 30 |
DENIED
|
moderate
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App\Entity\EntryComment {#2109
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|
Show voter details
|
| 31 |
DENIED
|
edit
|
App\Entity\EntryComment {#2109
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|
Show voter details
|
| 32 |
DENIED
|
moderate
|
App\Entity\EntryComment {#2109
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| 36 |
DENIED
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| 37 |
DENIED
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Show voter details
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| 38 |
DENIED
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moderate
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| 39 |
DENIED
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edit
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App\Entity\EntryComment {#2120
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Show voter details
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| 40 |
DENIED
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moderate
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App\Entity\EntryComment {#2120
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Show voter details
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| 41 |
DENIED
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ROLE_USER
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Show voter details
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| 42 |
DENIED
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moderate
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Proxies\__CG__\App\Entity\Entry {#2320
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Show voter details
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| 44 |
DENIED
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moderate
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| 45 |
DENIED
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ROLE_USER
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Show voter details
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| 46 |
DENIED
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moderate
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| 47 |
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Show voter details
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| 48 |
DENIED
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edit
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| 60 |
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App\Entity\EntryComment {#2050
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| 61 |
DENIED
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| 62 |
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moderate
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Proxies\__CG__\App\Entity\Entry {#2069
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Edit: this question has been answered now. Thank you to everyone who took the time to help me understand.\n
\n
> the premise that race is not a natural, biologically grounded feature of physically distinct subgroups of human beings but a socially constructed (culturally invented) category that is used to oppress and exploit people of colour.\n
\n
Okay… But we can take a DNA test and get our ancestry, telling us what percentage of what races make up our overall ethnicity. So how is race a social construct and not a biological feature, when we have a scientific method to determine our race? This part of the philosophy has been bothering me ever since I read it, and I’ve been hesitant to ask because of how offensive people get when you question this system.
"""
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Show voter details
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| 63 |
DENIED
|
edit
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Proxies\__CG__\App\Entity\Entry {#2069
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Edit: this question has been answered now. Thank you to everyone who took the time to help me understand.\n
\n
> the premise that race is not a natural, biologically grounded feature of physically distinct subgroups of human beings but a socially constructed (culturally invented) category that is used to oppress and exploit people of colour.\n
\n
Okay… But we can take a DNA test and get our ancestry, telling us what percentage of what races make up our overall ethnicity. So how is race a social construct and not a biological feature, when we have a scientific method to determine our race? This part of the philosophy has been bothering me ever since I read it, and I’ve been hesitant to ask because of how offensive people get when you question this system.
"""
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Show voter details
|
| 64 |
DENIED
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moderate
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Proxies\__CG__\App\Entity\Entry {#2069
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Edit: this question has been answered now. Thank you to everyone who took the time to help me understand.\n
\n
> the premise that race is not a natural, biologically grounded feature of physically distinct subgroups of human beings but a socially constructed (culturally invented) category that is used to oppress and exploit people of colour.\n
\n
Okay… But we can take a DNA test and get our ancestry, telling us what percentage of what races make up our overall ethnicity. So how is race a social construct and not a biological feature, when we have a scientific method to determine our race? This part of the philosophy has been bothering me ever since I read it, and I’ve been hesitant to ask because of how offensive people get when you question this system.
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Show voter details
|
| 65 |
DENIED
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ROLE_USER
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null |
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Show voter details
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| 66 |
DENIED
|
moderate
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App\Entity\EntryComment {#2072
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Edit: this question has been answered now. Thank you to everyone who took the time to help me understand.\n
\n
> the premise that race is not a natural, biologically grounded feature of physically distinct subgroups of human beings but a socially constructed (culturally invented) category that is used to oppress and exploit people of colour.\n
\n
Okay… But we can take a DNA test and get our ancestry, telling us what percentage of what races make up our overall ethnicity. So how is race a social construct and not a biological feature, when we have a scientific method to determine our race? This part of the philosophy has been bothering me ever since I read it, and I’ve been hesitant to ask because of how offensive people get when you question this system.
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Show voter details
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| 67 |
DENIED
|
edit
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App\Entity\EntryComment {#2072
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Edit: this question has been answered now. Thank you to everyone who took the time to help me understand.\n
\n
> the premise that race is not a natural, biologically grounded feature of physically distinct subgroups of human beings but a socially constructed (culturally invented) category that is used to oppress and exploit people of colour.\n
\n
Okay… But we can take a DNA test and get our ancestry, telling us what percentage of what races make up our overall ethnicity. So how is race a social construct and not a biological feature, when we have a scientific method to determine our race? This part of the philosophy has been bothering me ever since I read it, and I’ve been hesitant to ask because of how offensive people get when you question this system.
"""
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Show voter details
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| 68 |
DENIED
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moderate
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App\Entity\EntryComment {#2072
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Edit: this question has been answered now. Thank you to everyone who took the time to help me understand.\n
\n
> the premise that race is not a natural, biologically grounded feature of physically distinct subgroups of human beings but a socially constructed (culturally invented) category that is used to oppress and exploit people of colour.\n
\n
Okay… But we can take a DNA test and get our ancestry, telling us what percentage of what races make up our overall ethnicity. So how is race a social construct and not a biological feature, when we have a scientific method to determine our race? This part of the philosophy has been bothering me ever since I read it, and I’ve been hesitant to ask because of how offensive people get when you question this system.
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Show voter details
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| 77 |
DENIED
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ROLE_USER
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null |
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Show voter details
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| 78 |
DENIED
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moderate
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Proxies\__CG__\App\Entity\Entry {#2349
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Show voter details
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| 79 |
DENIED
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edit
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Proxies\__CG__\App\Entity\Entry {#2349
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Show voter details
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| 80 |
DENIED
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moderate
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Proxies\__CG__\App\Entity\Entry {#2349
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Show voter details
|
| 81 |
DENIED
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ROLE_USER
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null |
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Show voter details
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| 82 |
DENIED
|
moderate
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App\Entity\EntryComment {#2344
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|
Show voter details
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| 83 |
DENIED
|
edit
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App\Entity\EntryComment {#2344
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|
Show voter details
|
| 84 |
DENIED
|
moderate
|
App\Entity\EntryComment {#2344
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|
Show voter details
|
| 85 |
DENIED
|
ROLE_USER
|
null |
|
Show voter details
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| 86 |
DENIED
|
moderate
|
Proxies\__CG__\App\Entity\Entry {#2098
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Show voter details
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Show voter details
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| 88 |
DENIED
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moderate
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Show voter details
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| 89 |
DENIED
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ROLE_USER
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null |
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Show voter details
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| 90 |
DENIED
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moderate
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Show voter details
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| 91 |
DENIED
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edit
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App\Entity\EntryComment {#2140
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| 93 |
DENIED
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ROLE_USER
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Show voter details
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Show voter details
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| 95 |
DENIED
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edit
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App\Entity\EntryComment {#2077
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Show voter details
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| 96 |
DENIED
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moderate
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App\Entity\EntryComment {#2077
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Show voter details
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| 97 |
DENIED
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ROLE_USER
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Show voter details
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| 98 |
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moderate
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Proxies\__CG__\App\Entity\Entry {#2031
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Show voter details
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| 101 |
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Show voter details
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| 103 |
DENIED
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edit
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App\Entity\EntryComment {#2035
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Show voter details
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| 104 |
DENIED
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moderate
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App\Entity\EntryComment {#2035
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Show voter details
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| 105 |
DENIED
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Show voter details
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| 106 |
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moderate
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Proxies\__CG__\App\Entity\Entry {#2023
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|
Show voter details
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| 107 |
DENIED
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edit
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Proxies\__CG__\App\Entity\Entry {#2023
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|
Show voter details
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| 108 |
DENIED
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moderate
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Proxies\__CG__\App\Entity\Entry {#2023
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…2
} |
|
Show voter details
|
| 109 |
DENIED
|
ROLE_USER
|
null |
|
Show voter details
|
| 110 |
DENIED
|
moderate
|
App\Entity\EntryComment {#2019
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} |
|
Show voter details
|
| 111 |
DENIED
|
edit
|
App\Entity\EntryComment {#2019
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|
Show voter details
|
| 112 |
DENIED
|
moderate
|
App\Entity\EntryComment {#2019
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|
Show voter details
|
| 113 |
DENIED
|
ROLE_USER
|
null |
|
Show voter details
|
| 114 |
DENIED
|
moderate
|
Proxies\__CG__\App\Entity\Entry {#2395
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+slug: "What-are-your-criteria-for-upvoting-downvoting"
+title: "What are your criteria for upvoting/downvoting?"
+url: null
+body: """
I hate that I always compare Lemmy to Reddit, but Reddit used to have (not sure if they still do) guidelines called “Reddiquette” that included guidelines about upvoting and downvoting. I don’t remember the specifics (and sending too much of my browser traffic to Reddit makes me feel dirty) but one of the guidelines was not to upvote/downvote on the basis of agreement/disagreement with the content.\n
\n
On Lemmy, I’m honestly a bit lax about upvoting and downvoting at all. (I’m trying to be better about it.) Buy when I do upvote/downvote, I try to do so on the basis of whether the comment/post “adds to” or “subtracts from” the community or conversation. I can disagree with one comment’s take on some subject but still upvote them if they’ve given me a more nuanced perspective on the issue. If they’re just parrotting well-known talking points and not being thoughtful with their posts, I may downvote them evren if I agree with their ultimate stance.\n
\n
I’m just mostly wondering how folks on Lemmy think about upvotes/downvotes and what implications that has for the content here.
"""
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…2
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Show voter details
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| 115 |
DENIED
|
edit
|
Proxies\__CG__\App\Entity\Entry {#2395
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+magazine: Proxies\__CG__\App\Entity\Magazine {#1550 …}
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+title: "What are your criteria for upvoting/downvoting?"
+url: null
+body: """
I hate that I always compare Lemmy to Reddit, but Reddit used to have (not sure if they still do) guidelines called “Reddiquette” that included guidelines about upvoting and downvoting. I don’t remember the specifics (and sending too much of my browser traffic to Reddit makes me feel dirty) but one of the guidelines was not to upvote/downvote on the basis of agreement/disagreement with the content.\n
\n
On Lemmy, I’m honestly a bit lax about upvoting and downvoting at all. (I’m trying to be better about it.) Buy when I do upvote/downvote, I try to do so on the basis of whether the comment/post “adds to” or “subtracts from” the community or conversation. I can disagree with one comment’s take on some subject but still upvote them if they’ve given me a more nuanced perspective on the issue. If they’re just parrotting well-known talking points and not being thoughtful with their posts, I may downvote them evren if I agree with their ultimate stance.\n
\n
I’m just mostly wondering how folks on Lemmy think about upvotes/downvotes and what implications that has for the content here.
"""
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…2
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|
Show voter details
|
| 116 |
DENIED
|
moderate
|
Proxies\__CG__\App\Entity\Entry {#2395
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+magazine: Proxies\__CG__\App\Entity\Magazine {#1550 …}
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+slug: "What-are-your-criteria-for-upvoting-downvoting"
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+url: null
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I hate that I always compare Lemmy to Reddit, but Reddit used to have (not sure if they still do) guidelines called “Reddiquette” that included guidelines about upvoting and downvoting. I don’t remember the specifics (and sending too much of my browser traffic to Reddit makes me feel dirty) but one of the guidelines was not to upvote/downvote on the basis of agreement/disagreement with the content.\n
\n
On Lemmy, I’m honestly a bit lax about upvoting and downvoting at all. (I’m trying to be better about it.) Buy when I do upvote/downvote, I try to do so on the basis of whether the comment/post “adds to” or “subtracts from” the community or conversation. I can disagree with one comment’s take on some subject but still upvote them if they’ve given me a more nuanced perspective on the issue. If they’re just parrotting well-known talking points and not being thoughtful with their posts, I may downvote them evren if I agree with their ultimate stance.\n
\n
I’m just mostly wondering how folks on Lemmy think about upvotes/downvotes and what implications that has for the content here.
"""
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…2
} |
|
Show voter details
|
| 117 |
DENIED
|
ROLE_USER
|
null |
|
Show voter details
|
| 118 |
DENIED
|
moderate
|
App\Entity\EntryComment {#2394
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+url: null
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I hate that I always compare Lemmy to Reddit, but Reddit used to have (not sure if they still do) guidelines called “Reddiquette” that included guidelines about upvoting and downvoting. I don’t remember the specifics (and sending too much of my browser traffic to Reddit makes me feel dirty) but one of the guidelines was not to upvote/downvote on the basis of agreement/disagreement with the content.\n
\n
On Lemmy, I’m honestly a bit lax about upvoting and downvoting at all. (I’m trying to be better about it.) Buy when I do upvote/downvote, I try to do so on the basis of whether the comment/post “adds to” or “subtracts from” the community or conversation. I can disagree with one comment’s take on some subject but still upvote them if they’ve given me a more nuanced perspective on the issue. If they’re just parrotting well-known talking points and not being thoughtful with their posts, I may downvote them evren if I agree with their ultimate stance.\n
\n
I’m just mostly wondering how folks on Lemmy think about upvotes/downvotes and what implications that has for the content here.
"""
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} |
|
Show voter details
|
| 119 |
DENIED
|
edit
|
App\Entity\EntryComment {#2394
+user: App\Entity\User {#260 …}
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+slug: "What-are-your-criteria-for-upvoting-downvoting"
+title: "What are your criteria for upvoting/downvoting?"
+url: null
+body: """
I hate that I always compare Lemmy to Reddit, but Reddit used to have (not sure if they still do) guidelines called “Reddiquette” that included guidelines about upvoting and downvoting. I don’t remember the specifics (and sending too much of my browser traffic to Reddit makes me feel dirty) but one of the guidelines was not to upvote/downvote on the basis of agreement/disagreement with the content.\n
\n
On Lemmy, I’m honestly a bit lax about upvoting and downvoting at all. (I’m trying to be better about it.) Buy when I do upvote/downvote, I try to do so on the basis of whether the comment/post “adds to” or “subtracts from” the community or conversation. I can disagree with one comment’s take on some subject but still upvote them if they’ve given me a more nuanced perspective on the issue. If they’re just parrotting well-known talking points and not being thoughtful with their posts, I may downvote them evren if I agree with their ultimate stance.\n
\n
I’m just mostly wondering how folks on Lemmy think about upvotes/downvotes and what implications that has for the content here.
"""
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date: 2023-11-10 19:16:14.0 +01:00
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} |
|
Show voter details
|
| 120 |
DENIED
|
moderate
|
App\Entity\EntryComment {#2394
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+body: """
I hate that I always compare Lemmy to Reddit, but Reddit used to have (not sure if they still do) guidelines called “Reddiquette” that included guidelines about upvoting and downvoting. I don’t remember the specifics (and sending too much of my browser traffic to Reddit makes me feel dirty) but one of the guidelines was not to upvote/downvote on the basis of agreement/disagreement with the content.\n
\n
On Lemmy, I’m honestly a bit lax about upvoting and downvoting at all. (I’m trying to be better about it.) Buy when I do upvote/downvote, I try to do so on the basis of whether the comment/post “adds to” or “subtracts from” the community or conversation. I can disagree with one comment’s take on some subject but still upvote them if they’ve given me a more nuanced perspective on the issue. If they’re just parrotting well-known talking points and not being thoughtful with their posts, I may downvote them evren if I agree with their ultimate stance.\n
\n
I’m just mostly wondering how folks on Lemmy think about upvotes/downvotes and what implications that has for the content here.
"""
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DENIED
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| 124 |
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moderate
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| 125 |
DENIED
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ROLE_USER
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Show voter details
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| 126 |
DENIED
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moderate
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Show voter details
|
| 127 |
DENIED
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edit
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Show voter details
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| 128 |
DENIED
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moderate
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moderate
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| 135 |
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edit
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App\Entity\EntryComment {#2422
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Show voter details
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| 136 |
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moderate
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App\Entity\EntryComment {#2422
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| 138 |
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moderate
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Proxies\__CG__\App\Entity\Entry {#1910
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Show voter details
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| 139 |
DENIED
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edit
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Proxies\__CG__\App\Entity\Entry {#1910
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Show voter details
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| 140 |
DENIED
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moderate
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Proxies\__CG__\App\Entity\Entry {#1910
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…2
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|
Show voter details
|
| 141 |
DENIED
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ROLE_USER
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null |
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Show voter details
|
| 142 |
DENIED
|
moderate
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App\Entity\EntryComment {#2383
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|
Show voter details
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| 143 |
DENIED
|
edit
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App\Entity\EntryComment {#2383
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|
Show voter details
|
| 144 |
DENIED
|
moderate
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App\Entity\EntryComment {#2383
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|
Show voter details
|
| 145 |
DENIED
|
ROLE_USER
|
null |
|
Show voter details
|
| 146 |
DENIED
|
moderate
|
Proxies\__CG__\App\Entity\Entry {#1724
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The Dicks also had a good position \n
<https://piped.video/watch?v=lUI2kZVR6W8>
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| 147 |
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edit
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Show voter details
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| 148 |
DENIED
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moderate
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Show voter details
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| 149 |
DENIED
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ROLE_USER
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Show voter details
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| 150 |
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moderate
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Show voter details
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| 151 |
DENIED
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edit
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App\Entity\EntryComment {#1729
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| 152 |
DENIED
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moderate
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App\Entity\EntryComment {#1729
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| 153 |
DENIED
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ROLE_USER
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Show voter details
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| 154 |
DENIED
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moderate
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Proxies\__CG__\App\Entity\Entry {#1597
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| 156 |
DENIED
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moderate
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Show voter details
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| 157 |
DENIED
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ROLE_USER
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null |
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Show voter details
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| 158 |
DENIED
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moderate
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App\Entity\EntryComment {#1625
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| 159 |
DENIED
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edit
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App\Entity\EntryComment {#1625
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Show voter details
|
| 160 |
DENIED
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moderate
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App\Entity\EntryComment {#1625
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Show voter details
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| 161 |
DENIED
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ROLE_USER
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null |
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Show voter details
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| 162 |
DENIED
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moderate
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Proxies\__CG__\App\Entity\Entry {#1683
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|
Show voter details
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| 163 |
DENIED
|
edit
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Proxies\__CG__\App\Entity\Entry {#1683
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|
Show voter details
|
| 164 |
DENIED
|
moderate
|
Proxies\__CG__\App\Entity\Entry {#1683
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…2
} |
|
Show voter details
|
| 165 |
DENIED
|
ROLE_USER
|
null |
|
Show voter details
|
| 166 |
DENIED
|
moderate
|
App\Entity\EntryComment {#1701
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|
Show voter details
|
| 167 |
DENIED
|
edit
|
App\Entity\EntryComment {#1701
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} |
|
Show voter details
|
| 168 |
DENIED
|
moderate
|
App\Entity\EntryComment {#1701
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|
Show voter details
|
| 169 |
DENIED
|
ROLE_USER
|
null |
|
Show voter details
|
| 170 |
DENIED
|
moderate
|
Proxies\__CG__\App\Entity\Entry {#1564
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+url: null
+body: """
I see a lot of posts lately, mainly in ‘world news’ communities, that when I investigate their source, I cannot come to any other conclostion that purposefully spreading of fake news and propaganda on lemmy.\n
\n
I love this platform and want to see it thrive, but the fact that these kind of posts can so easily populate my feed is disturbing.
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…2
} |
|
Show voter details
|
| 171 |
DENIED
|
edit
|
Proxies\__CG__\App\Entity\Entry {#1564
+user: Proxies\__CG__\App\Entity\User {#2851 …}
+magazine: Proxies\__CG__\App\Entity\Magazine {#1550 …}
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+url: null
+body: """
I see a lot of posts lately, mainly in ‘world news’ communities, that when I investigate their source, I cannot come to any other conclostion that purposefully spreading of fake news and propaganda on lemmy.\n
\n
I love this platform and want to see it thrive, but the fact that these kind of posts can so easily populate my feed is disturbing.
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Show voter details
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| 172 |
DENIED
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moderate
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Proxies\__CG__\App\Entity\Entry {#1564
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Show voter details
|
| 173 |
DENIED
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ROLE_USER
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null |
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Show voter details
|
| 174 |
DENIED
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moderate
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App\Entity\EntryComment {#1563
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|
Show voter details
|
| 175 |
DENIED
|
edit
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App\Entity\EntryComment {#1563
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I see a lot of posts lately, mainly in ‘world news’ communities, that when I investigate their source, I cannot come to any other conclostion that purposefully spreading of fake news and propaganda on lemmy.\n
\n
I love this platform and want to see it thrive, but the fact that these kind of posts can so easily populate my feed is disturbing.
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|
Show voter details
|
| 176 |
DENIED
|
moderate
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App\Entity\EntryComment {#1563
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I see a lot of posts lately, mainly in ‘world news’ communities, that when I investigate their source, I cannot come to any other conclostion that purposefully spreading of fake news and propaganda on lemmy.\n
\n
I love this platform and want to see it thrive, but the fact that these kind of posts can so easily populate my feed is disturbing.
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|
Show voter details
|
| 177 |
DENIED
|
ROLE_USER
|
null |
|
Show voter details
|
| 178 |
DENIED
|
moderate
|
Proxies\__CG__\App\Entity\Entry {#1577
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|
Show voter details
|
| 179 |
DENIED
|
edit
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Proxies\__CG__\App\Entity\Entry {#1577
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| 181 |
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| 183 |
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App\Entity\EntryComment {#1615
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| 184 |
DENIED
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